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Judul Periodontitis : From Dysbiotic Microbial Immune Response to Systemic Inflammation / Anders Johansson (editor)
Pengarang Johansson, Anders
Penerbitan Basel : MDPI - Multidisciplinary Digital Publishing Institute, 2020
Deskripsi Fisik 122 p. :ill.
ISBN 978-3-03943-508-1
Subjek PERIODONTOLOGY
Catatan Periodontitis is an infection-induced inflammatory disease accounting for huge healthcare costs and socio-economic impacts. Bacteria from the indigenous oral flora colonize the interspace between the tooth and the connective tissue, which induces an inflammatory response. If the bacteria proliferate and release virulence factors, they cause an imbalance in the host inflammatory response that induces degenerative processes in the surrounding tissues. This process is often slow, and the disease affects mainly older people, but the process could be rapid and affect young individuals if certain pathogens colonize the area. The two most studied periodontal pathogens, Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans, express virulence factors, including proteases and exotoxins. Periodontal bacteria and their products can be translocated to the peripheral circulation and are therefore linked to the risk pattern of several systemic diseases. However, it is not known if the increased risk for systemi
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Lokasi Akses Online https://directory.doabooks.org/handle/20.500.12854/69297

 
No Barcode No. Panggil Akses Lokasi Ketersediaan
168425192 617.6 Joh p Baca di tempat Perpustakaan Pusat - Online Resources
Ebook
Tersedia
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100 1 # $a Johansson, Anders
245 1 # $a Periodontitis : $b From Dysbiotic Microbial Immune Response to Systemic Inflammation /$c Anders Johansson (editor)
260 # # $a Basel :$b MDPI - Multidisciplinary Digital Publishing Institute,$c 2020
300 # # $a 122 p. : $b ill.
505 # # $a Periodontitis is an infection-induced inflammatory disease accounting for huge healthcare costs and socio-economic impacts. Bacteria from the indigenous oral flora colonize the interspace between the tooth and the connective tissue, which induces an inflammatory response. If the bacteria proliferate and release virulence factors, they cause an imbalance in the host inflammatory response that induces degenerative processes in the surrounding tissues. This process is often slow, and the disease affects mainly older people, but the process could be rapid and affect young individuals if certain pathogens colonize the area. The two most studied periodontal pathogens, Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans, express virulence factors, including proteases and exotoxins. Periodontal bacteria and their products can be translocated to the peripheral circulation and are therefore linked to the risk pattern of several systemic diseases. However, it is not known if the increased risk for systemic disease associated with periodontitis is an effect of the invading bacteria and/or their released products, the release of components from the local inflammatory response, or a common host susceptibility pattern. The most studied periodontitis-associated systemic diseases are cardiovascular diseases and rheumatoid arthritis. Here, we want to shed light on mechanisms behind the associations of periodontal infections with systemic inflammation.
650 # # $a PERIODONTOLOGY
856 # # $a https://directory.doabooks.org/handle/20.500.12854/69297
990 # # $a 168425192
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